Since experimental fracture tests on endodontically treated teeth with porcelain restorations show that even in fracture, the dentin bond survives and the tooth breaks instead, in what way do you think this may alter the dynamics of biologic width invasion or crown lengthening procedures?

And if a bonded margin is subgingival (and therefore no marginal gap, assuming isolation during the bonding), and there is no microenvironment for bacterial colonization, does that alter our perception of biologic width compromise as it does with a cemented crown with it's inherent marginal gap?

I am interested in the thoughts of those on this forum regarding this important subject that may effect the way we all practice dentistry and may alter the current concepts of biologic width compromise and treatment.